By M. Chen, T. Pope, D. Ott(eds0
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This case (Fig. 3â 44) is an example of a long-standing hypertension in a patient with shortness of breath and pulmonary edema from congestive heart failure. ) Note the increased size of the cardiac silhouette, the ill-defined reticular perihilar opacities, and the redistribution of blood flow to the upper lung zones. In this woman, the cause of the pulmonary edema was myocardial infarction. Discussion: The main pulmonary arteries are large, the lobar arteries smaller, and each branching segment becomes progressively smaller.
This positioning may cause the pacemaker to function poorly or cause pleural and/or pericardial effusion. Because the leads are introduced via the subclavian vein, pneumothorax is also a potential complication. ) A CT confirmed the pacemaker lead perforation (Fig. 3â 65). 3-30. The patient in this case (Fig. 3â 64) had hypoplastic left heart syndrome as an infant.
The most likely diagnosis in Case 3-2 (Fig. 3â 25) is A. mediastinal masses. B. intracardiac shunts (ASD and VSD). C. pericardial effusion or cardiomyopathy. D. combined aortic and pulmonary arterial disease. E. technical aberrations. 3-3. The most likely diagnosis in Case 3-3 (Fig. 3â 26) is A. mediastinal masses. B. intracardiac shunts (ASD and VSD). C. pericardial effusion or cadiomyopathy. D. combined aortic and pulmonary arterial disease. E. technical aberrations. 3-4. The most likely diagnosis in Case 3-4 (Fig.
Basic Radiology by M. Chen, T. Pope, D. Ott(eds0